Journal article
K. Margolis, J. Vittorio, M. Talavera, Karen Gluck, Zhishan Li, A. Iuga, Korey D. Stevanovic, Virginia Saurman, Narek Israelyan, M. Welch, M. Gershon
2017
2017
Semantic Scholar
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Cite
APA
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Margolis, K., Vittorio, J., Talavera, M., Gluck, K., Li, Z., Iuga, A., … Gershon, M. (2017). Hormones, Neurotransmitters, Growth Factors, Receptors, and Signaling Enteric serotonin and oxytocin: endogenous regulation of severity in a murine model of necrotizing enterocolitis.
Chicago/Turabian
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Margolis, K., J. Vittorio, M. Talavera, Karen Gluck, Zhishan Li, A. Iuga, Korey D. Stevanovic, et al. “Hormones, Neurotransmitters, Growth Factors, Receptors, and Signaling Enteric Serotonin and Oxytocin: Endogenous Regulation of Severity in a Murine Model of Necrotizing Enterocolitis” (2017).
MLA
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Margolis, K., et al. Hormones, Neurotransmitters, Growth Factors, Receptors, and Signaling Enteric Serotonin and Oxytocin: Endogenous Regulation of Severity in a Murine Model of Necrotizing Enterocolitis. 2017.
BibTeX Click to copy
@article{k2017a,
title = {Hormones, Neurotransmitters, Growth Factors, Receptors, and Signaling Enteric serotonin and oxytocin: endogenous regulation of severity in a murine model of necrotizing enterocolitis},
year = {2017},
author = {Margolis, K. and Vittorio, J. and Talavera, M. and Gluck, Karen and Li, Zhishan and Iuga, A. and Stevanovic, Korey D. and Saurman, Virginia and Israelyan, Narek and Welch, M. and Gershon, M.}
}
Abstract
Enteric serotonin and oxytocin: endogenous regulation of severity in a murine model of necrotizing enterocolitis. Am J Physiol Gastrointest Liver Physiol 313: G386–G398, 2017. enterocolitis (NEC), a gastrointestinal inflammatory disease of unknown etiology that may also affect the liver, causes a great deal of morbidity and mortality in premature infants. We tested the hypothesis that signaling molecules, which are endogenous to the bowel, regulate the severity of intestinal and hepatic damage in an established murine NEC model. Specifically, we postulated that mucosal serotonin (5-HT), which is proinflammatory, would exacerbate experimental NEC and that oxytocin (OT), which is present in enteric neurons and is
